All terms in GO
| Label | Id | Description |
|---|---|---|
| negative regulation of intrinsic apoptotic signaling pathway in response to osmotic stress | GO_1902219 | [Any process that stops, prevents or reduces the frequency, rate or extent of intrinsic apoptotic signaling pathway in response to osmotic stress.] |
| positive regulation of intrinsic apoptotic signaling pathway in response to osmotic stress by p53 class mediator | GO_1902240 | [Any process that activates or increases the frequency, rate or extent of intrinsic apoptotic signaling pathway in response to osmotic stress by p53 class mediator.] |
| positive regulation of intrinsic apoptotic signaling pathway in response to osmotic stress | GO_1902220 | [Any process that activates or increases the frequency, rate or extent of intrinsic apoptotic signaling pathway in response to osmotic stress.] |
| copal-8-ol diphosphate(3-) metabolic process | GO_1902241 | [The chemical reactions and pathways involving copal-8-ol diphosphate(3-).] |
| copal-8-ol diphosphate(3-) catabolic process | GO_1902242 | [The chemical reactions and pathways resulting in the breakdown of copal-8-ol diphosphate(3-).] |
| copal-8-ol diphosphate(3-) biosynthetic process | GO_1902243 | [The chemical reactions and pathways resulting in the formation of copal-8-ol diphosphate(3-).] |
| cis-abienol metabolic process | GO_1902244 | [The chemical reactions and pathways involving cis-abienol.] |
| cis-abienol catabolic process | GO_1902245 | [The chemical reactions and pathways resulting in the breakdown of cis-abienol.] |
| cis-abienol biosynthetic process | GO_1902246 | [The chemical reactions and pathways resulting in the formation of cis-abienol.] |
| geranylgeranyl diphosphate catabolic process | GO_1902247 | [The chemical reactions and pathways resulting in the breakdown of geranylgeranyl diphosphate.] |
| geranylgeranyl diphosphate metabolic process | GO_0033385 | [The chemical reactions and pathways involving geranylgeranyl diphosphate, a polyprenol compound involved in the biosynthesis of a variety of terpenoids including chlorophylls, carotenoids, tocopherols, plastoquinones, and the plant hormones gibberellins.] |
| terpenoid catabolic process | GO_0016115 | [The chemical reactions and pathways resulting in the breakdown of terpenoids, any member of a class of compounds characterized by an isoprenoid chemical structure.] |
| 5-O-phosphono-alpha-D-ribofuranosyl diphosphate binding | GO_1902248 | [Interacting selectively and non-covalently with 5-O-phosphono-alpha-D-ribofuranosyl diphosphate.] |
| IMP binding | GO_1902249 | [Interacting selectively and non-covalently with IMP, inosine monophosphate.] |
| purine ribonucleotide binding | GO_0032555 | [Interacting selectively and non-covalently with a purine ribonucleotide, any compound consisting of a purine ribonucleoside that is esterified with (ortho)phosphate or an oligophosphate at any hydroxyl group on the ribose moiety.] |
| receptor localization to non-motile cilium | GO_0097500 | [A process in which a receptor is transported to, or maintained in, a location within a non-motile cilium.] |
| protein localization to ciliary membrane | GO_1903441 | [A process in which a protein is transported to, or maintained in, a location within a ciliary membrane.] |
| protein localization to non-motile cilium | GO_0097499 | [A process in which a protein is transported to, or maintained in, a location within a non-motile cilium.] |
| mannosylation | GO_0097502 | [The covalent attachment of a mannose residue to a substrate molecule.] |
| Gemini of coiled bodies | GO_0097504 | [Nuclear bodies frequently found near or associated with Cajal bodies (also called coiled bodies or CBs). Gemini of coiled bodies, or 'gems', are similar in size and shape to CBs, and often indistinguishable under the microscope. Unlike CBs, gems do not contain small nuclear ribonucleoproteins (snRNPs); they contain a protein called survivor of motor neurons (SMN) whose function relates to snRNP biogenesis. Gems are believed to assist CBs in snRNP biogenesis, and to play a role in the etiology of spinal muscular atrophy (SMA).] |