All terms in GO
| Label | Id | Description |
|---|---|---|
| positive regulation of tyramine signaling pathway | GO_2000133 | [Any process that activates or increases the frequency, rate or extent of tyramine signaling pathway.] |
| negative regulation of cell cycle G1/S phase transition | GO_1902807 | [Any signalling pathway that decreases or inhibits the activity of a cell cycle cyclin-dependent protein kinase to modulate the switch from G1 phase to S phase of the cell cycle.] |
| obsolete Rieske iron-sulfur protein | GO_0000047 | [OBSOLETE. (Was not defined before being made obsolete).] |
| peptidyltransferase activity | GO_0000048 | [Catalysis of the reaction: peptidyl-tRNA(1) + aminoacyl-tRNA(2) = tRNA(1) + peptidylaminoacyl-tRNA(2).] |
| autophagosome organization | GO_1905037 | [A process that is carried out at the cellular level which results in the assembly, arrangement of constituent parts, or disassembly of an autophagosome.] |
| Atg12 conjugating enzyme activity | GO_0061651 | [Isoenergetic transfer of Atg12 from one protein to another via the reaction X-Atg12 + Y -> Y-Atg12 + X, where both the X-Atg12 and Y-Atg12 linkages are thioester bonds between the C-terminal amino acid of Atg12 and a sulfhydryl side group of a cysteine residue.] |
| Atg12 ligase activity | GO_0061660 | [Catalysis of the transfer of Atg12 to a substrate protein via the reaction X-Atg12 + S --> X + S-Atg12, where X is either an E2 or E3 enzyme, the X-Atg12 linkage is a thioester bond, and the S-Atg12 linkage is an isopeptide bond between the C-terminal amino acid of Atg12 and the epsilon-amino group of lysine residues in the substrate.] |
| GO_0000046 | GO_0000046 | |
| GO_0000043 | GO_0000043 | |
| obsolete ascorbate stabilization | GO_0000044 | [OBSOLETE. The reduction of the ascorbate free radical to a stable form.] |
| regulation of response to food | GO_0032095 | [Any process that modulates the frequency, rate or extent of a response to a food stimulus.] |
| transition metal ion transport | GO_0000041 | [The directed movement of transition metal ions into, out of or within a cell, or between cells, by means of some agent such as a transporter or pore. A transition metal is an element whose atom has an incomplete d-subshell of extranuclear electrons, or which gives rise to a cation or cations with an incomplete d-subshell. Transition metals often have more than one valency state. Biologically relevant transition metals include vanadium, manganese, iron, copper, cobalt, nickel, molybdenum and silver.] |
| GO_0000042 | GO_0000042 | |
| negative regulation of cell proliferation involved in heart morphogenesis | GO_2000137 | [Any process that stops, prevents, or reduces the frequency, rate or extent of cell proliferation involved in heart morphogenesis.] |
| positive regulation of cell proliferation involved in heart morphogenesis | GO_2000138 | [Any process that activates or increases the frequency, rate or extent of cell proliferation involved in heart morphogenesis.] |
| GO_0000040 | GO_0000040 | |
| negative regulation of mammary stem cell proliferation | GO_2000102 | [Any process that stops, prevents, or reduces the frequency, rate or extent of mammary stem cell proliferation.] |
| regulation of mammary stem cell proliferation | GO_2000101 | [Any process that modulates the frequency, rate or extent of mammary stem cell proliferation.] |
| mammary stem cell proliferation | GO_0002174 | [The expansion of a mammary stem cell population by cell division. Mammary stem cells are a source of cells for growth of the mammary gland during puberty and gestation. These cells can give rise to both the luminal and myoepithelial cell types of the gland, and can regenerate the entire organ.] |
| positive regulation of mammary stem cell proliferation | GO_2000103 | [Any process that activates or increases the frequency, rate or extent of mammary stem cell proliferation.] |