All terms in GO
| Label | Id | Description |
|---|---|---|
| protein guanylylation | GO_0018260 | [The addition of phospho-guanosine to a protein amino acid.] |
| anaerobic L-alanine catabolic process | GO_0019667 | [The anaerobic chemical reactions and pathways resulting in the breakdown of L-alanine, yielding energy in the form of ATP.] |
| anaerobic catabolism of pairs of amino acids | GO_0019668 | [The anaerobic chemical reactions and pathways resulting in the breakdown of amino acids; in these reactions, one amino acid is oxidised (acts as an electron donor) and a different amino acid is reduced (acts as an electron acceptor); oxidation of the electron-donating amino acid yields energy in the form of ATP.] |
| anaerobic glycine catabolic process | GO_0019669 | [The anaerobic chemical reactions and pathways resulting in the breakdown of glycine, yielding energy in the form of ATP.] |
| neurotrophin receptor activity | GO_0005030 | [Combining with a neurotrophin, any of a family of growth factors that prevent apoptosis in neurons and promote nerve growth, and transmitting the signal to initiate a change in cell activity.] |
| signaling receptor activity | GO_0038023 | [Receiving a signal and transmitting it in the cell to initiate a change in cell activity. A signal is a physical entity or change in state that is used to transfer information in order to trigger a response.] |
| neurotrophin binding | GO_0043121 | [Interacting selectively and non-covalently with a neurotrophin, any of a family of growth factors that prevent apoptosis in neurons and promote nerve growth.] |
| neurotrophin signaling pathway | GO_0038179 | [A series of molecular signals initiated by the binding of a neurotrophin to a receptor on the surface of a target cell, and ending with regulation of a downstream cellular process, e.g. transcription. Neurotrophins are a family of secreted growth factors that induce the survival, development, and function of neurons.] |
| tumor necrosis factor-activated receptor activity | GO_0005031 | [Combining with tumor necrosis factor, a proinflammatory cytokine produced by monocytes and macrophages, to initiate a change in cell function.] |
| death receptor activity | GO_0005035 | [Combining with an extracellular messenger (called a death ligand), and transmitting the signal from one side of the plasma membrane to the other to initiate apoptotic or necrotic cell death.] |
| tumor necrosis factor binding | GO_0043120 | [Interacting selectively and non-covalently with tumor necrosis factor, a proinflammatory cytokine produced by monocytes and macrophages.] |
| tumor necrosis factor-mediated signaling pathway | GO_0033209 | [A series of molecular signals initiated by the binding of a tumor necrosis factor to a receptor on the surface of a cell, and ending with regulation of a downstream cellular process, e.g. transcription.] |
| GO_0005032 | GO_0005032 | |
| GO_0005033 | GO_0005033 | |
| osmosensor activity | GO_0005034 | [Sensing extracellular osmolarity to initiate a change in cell activity, and spanning the membrane of the cell.] |
| programmed cell death | GO_0012501 | [A process which begins when a cell receives an internal or external signal and activates a series of biochemical events (signaling pathway). The process ends with the death of the cell.] |
| obsolete death receptor adaptor protein activity | GO_0005037 | [OBSOLETE. (Was not defined before being made obsolete).] |
| negative regulation of complement activation, alternative pathway in other organism | GO_0044634 | [Any process that stops, prevents, or reduces the frequency, rate or extent of complement activation by the alternative pathway, in a different organism.] |
| modulation of complement activation, alternative pathway in other organism | GO_0044633 | [A process that modulates the frequency, rate or extent of complement activation, via the alternative pathway, in a different organism.] |
| negative regulation of complement activation, alternative pathway | GO_0045957 | [Any process that stops, prevents, or reduces the frequency, rate or extent of complement activation by the alternative pathway.] |