All terms in GO
| Label | Id | Description |
|---|---|---|
| transcription antitermination | GO_0031564 | [Regulation of transcription by a mechanism that allows RNA polymerase to continue transcription beyond termination site(s).] |
| obsolete cytokinesis checkpoint | GO_0031565 | [OBSOLETE. A mitotic cell cycle checkpoint that detects a defect in cytokinesis and negatively regulates G2/M transition.] |
| actomyosin contractile ring maintenance | GO_0031566 | [The cell cycle process in which the contractile ring is maintained in response to the cytokinesis checkpoint; that is when cytokinesis is delayed awaiting completion of nuclear division or the correct formation of cytokinetic structures. This process occurs in the context of cytokinesis that takes place as part of a cell cycle.] |
| contractile ring maintenance | GO_0036212 | [The process in which the contractile ring is maintained, typically in response to an internal or external cue.] |
| mitotic cell size control checkpoint | GO_0031567 | [The mitotic cell cycle checkpoint that delays or arrests cell cycle progression until cells have reached a critical size.] |
| regulation of cell size | GO_0008361 | [Any process that modulates the size of a cell.] |
| G1 cell size control checkpoint | GO_0031568 | [A mitotic cell cycle checkpoint that monitors cell size, and negatively regulates cell cycle progression between G1 and S phase until a critical size is reached.] |
| mitotic G2 cell size control checkpoint | GO_0031569 | [A mitotic cell cycle checkpoint that monitors cell size, and negatively regulates cell cycle progression between G2 and M phase until a critical size is reached.] |
| positive regulation of brain-derived neurotrophic factor receptor signaling pathway | GO_0031550 | [Any process that activates or increases the frequency, rate or extent of signaling via the brain-derived neurotrophic factor receptor signaling pathway.] |
| regulation of brain-derived neurotrophic factor-activated receptor activity | GO_0031551 | [Any process that modulates the frequency, rate or extent of brain-derived neurotrophic factor-activated receptor activity.] |
| regulation of cellular response to growth factor stimulus | GO_0090287 | [Any process that modulates the rate, frequency, or extent of a change in state or activity of a cell (in terms of movement, secretion, enzyme production, gene expression, etc.) as a result of a growth factor stimulus.] |
| regulation of protein tyrosine kinase activity | GO_0061097 | [Any process that modulates the rate, frequency, or extent of protein tyrosine kinase activity.] |
| brain-derived neurotrophic factor-activated receptor activity | GO_0060175 | [Combining with a brain-derived neurotrophic factor and transmitting the signal across the plasma membrane to initiate a change in cell activity.] |
| negative regulation of brain-derived neurotrophic factor-activated receptor activity | GO_0031552 | [Any process that stops, prevents, or reduces the frequency, rate or extent of brain-derived neurotrophic factor-activated receptor activity.] |
| negative regulation of cellular response to growth factor stimulus | GO_0090288 | [Any process that decreases the rate, frequency, or extent of a change in state or activity of a cell (in terms of movement, secretion, enzyme production, gene expression, etc.) as a result of a growth factor stimulus.] |
| positive regulation of brain-derived neurotrophic factor-activated receptor activity | GO_0031553 | [Any process that activates or increases the frequency, rate or extent of brain-derived neurotrophic factor-activated receptor activity.] |
| DNA-templated transcription, termination | GO_0006353 | [The cellular process that completes DNA-templated transcription; the formation of phosphodiester bonds ceases, the RNA-DNA hybrid dissociates, and RNA polymerase releases the DNA.] |
| transcriptional attenuation | GO_0031555 | [Regulation of transcription through variation in where transcription termination occurs.] |
| transcriptional attenuation by ribosome | GO_0031556 | [A type of transcriptional regulation at the level of early termination. This process can occur only in prokaryotes, where transcription of an operon into messenger RNA and translation of that mRNA into polypeptides occur simultaneously. The general principle is that alternative mRNA secondary structures occur under different physiological conditions such as available amount of a particular amino acid. One set of conditions favors early termination of transcription. In the classic example of the trp biosynthesis operon, translation of the gene for a short, trp-containing polypeptide called the trp operon leader peptide pauses either at a trp codon (if tryptophan is scarce) or the stop codon (if trp is readily available). In the former situation transcription continues, but in the latter a Rho-independent terminator forms and reduces, or attenuates, expression of the tryptophan biosynthesis genes. Although the polypeptides encoded by leader peptide genes appear not to be stable once their translation is complete, it is suggested by recent studies that their nascent polypeptide chains interact specifically with ribosomes, specific uncharged tRNAs, or other cellular components to inhibit release at the stop codon and improve the function of transcriptional attenuation as a regulatory switch.] |
| obsolete induction of programmed cell death in response to chemical stimulus | GO_0031557 | [OBSOLETE. A process which directly activates any of the steps required for programmed cell death as a result of a chemical stimulus.] |