All terms in MESH
| Label | Id | Description |
|---|---|---|
| Oxidopamine | D016627 | [A neurotransmitter analogue that depletes noradrenergic stores in nerve endings and induces a reduction of dopamine levels in the brain. Its mechanism of action is related to the production of cytolytic free-radicals.] |
| Hydroxydopamines | D006892 | [Dopamines with a hydroxy group substituted in one or more positions.] |
| Replica Techniques | D016628 | [Methods of preparing tissue specimens for visualization using an electron microscope, usually a scanning electron microscope. The methods involve the creation of exact copies of the specimens by making a mold or cast (i.e., replica) of the specimen.] |
| Histocytological Preparation Techniques | D016591 | [Methods of preparing cells or tissues for examination and study of their origin, structure, function, or pathology. The methods include preservation, fixation, sectioning, staining, replica, or other technique to allow for viewing using a microscope.] |
| Esophagectomy | D016629 | [Excision of part (partial) or all (total) of the esophagus. (Dorland, 28th ed)] |
| 3,5-methanoproline | C471505 | |
| deethylatrazine | C446541 | |
| Corneal Neovascularization | D016510 | [New blood vessels originating from the corneal veins and extending from the limbus into the adjacent CORNEAL STROMA. Neovascularization in the superficial and/or deep corneal stroma is a sequel to numerous inflammatory diseases of the ocular anterior segment, such as TRACHOMA, viral interstitial KERATITIS, microbial KERATOCONJUNCTIVITIS, and the immune response elicited by CORNEAL TRANSPLANTATION.] |
| Corneal Diseases | D003316 | [Diseases of the cornea.] |
| Severe Combined Immunodeficiency | D016511 | [Group of rare congenital disorders characterized by impairment of both humoral and cell-mediated immunity, leukopenia, and low or absent antibody levels. It is inherited as an X-linked or autosomal recessive defect. Mutations occurring in many different genes cause human Severe Combined Immunodeficiency (SCID).] |
| Infant, Newborn, Diseases | D007232 | [Diseases of newborn infants present at birth (congenital) or developing within the first month of birth. It does not include hereditary diseases not manifesting at birth or within the first 30 days of life nor does it include inborn errors of metabolism. Both HEREDITARY DISEASES and METABOLISM, INBORN ERRORS are available as general concepts.] |
| Immunologic Deficiency Syndromes | D007153 | [Syndromes in which there is a deficiency or defect in the mechanisms of immunity, either cellular or humoral.] |
| Ankle Injuries | D016512 | [Harm or hurt to the ankle or ankle joint usually inflicted by an external source.] |
| Leg Injuries | D007869 | [General or unspecified injuries involving the leg.] |
| Mice, SCID | D016513 | [Mice homozygous for the mutant autosomal recessive gene "scid" which is located on the centromeric end of chromosome 16. These mice lack mature, functional lymphocytes and are thus highly susceptible to lethal opportunistic infections if not chronically treated with antibiotics. The lack of B- and T-cell immunity resembles severe combined immunodeficiency (SCID) syndrome in human infants. SCID mice are useful as animal models since they are receptive to implantation of a human immune system producing SCID-human (SCID-hu) hematochimeric mice.] |
| Mice, Mutant Strains | D008817 | [Mice bearing mutant genes which are phenotypically expressed in the animals.] |
| Genes, Neurofibromatosis 1 | D016514 | [Tumor suppressor genes located on the long arm of human chromosome 17 in the region 17q11.2. Mutation of these genes is thought to cause NEUROFIBROMATOSIS 1, Watson syndrome, and LEOPARD syndrome.] |
| Genes, Tumor Suppressor | D016147 | [Genes that inhibit expression of the tumorigenic phenotype. They are normally involved in holding cellular growth in check. When tumor suppressor genes are inactivated or lost, a barrier to normal proliferation is removed and unregulated growth is possible.] |
| Genes, Neurofibromatosis 2 | D016515 | [Tumor suppressor genes located on the long arm of human chromosome 22. Mutation or loss of these genes causes NEUROFIBROMATOSIS 2.] |
| CD4-CD8 Ratio | D016516 | [Ratio of T-LYMPHOCYTES that express the CD4 ANTIGEN to those that express the CD8 ANTIGEN. This value is commonly assessed in the diagnosis and staging of diseases affecting the IMMUNE SYSTEM including HIV INFECTIONS.] |