All terms in NCIT
| Label | Id | Description |
|---|---|---|
| Biphasic Pattern | NCIT_C35907 | |
| Monophasic Pattern | NCIT_C35908 | |
| Pseudofollicular Pattern | NCIT_C35901 | |
| Interfollicular Pattern | NCIT_C35902 | |
| Diffuse Pattern | NCIT_C35903 | [A morphologic architectural pattern in which a cellular population is distributed in a relatively uniform fashion throughout a specified tissue area; not focal.] |
| Nodular and Diffuse Pattern | NCIT_C35904 | |
| Multinodular Pattern | NCIT_C35900 | |
| Start Procedure Action Item | NCIT_C69263 | [Indicates the beginning action of a procedure.] |
| End Procedure Action Item | NCIT_C69264 | [Indicates the ending action of a procedure.] |
| Image Acquired | NCIT_C69265 | [An indication that an image has been successfully captured and saved.] |
| Acquisition | NCIT_C43384 | [The process by which something is acquired or obtained.] |
| Fibromun | NCIT_C94210 | [An immunocytokine consisting of human pro-inflammatory cytokine tumor necrosis factor alpha (TNFalpha) fused to a human single-chain variable fragment (scFv) directed against the extra-domain B (ED-B) of fibronectin (L19), with potential immunopotentiating and antineoplastic activities. The L19 moiety of recombinant human fusion protein L19TNFalpha binds to the ED-B domain of fibronectin on tumor cells in the tumor neovasculature. In turn, the TNFalpha moiety may locally induce an immune response against ED-B fibronectin-expressing tumor cells and may specifically decrease the proliferation of ED-B-expressing tumor cells. ED-B is predominantly expressed during angiogenesis and tumor growth.] |
| Glioblastoma Multiforme Multipeptide Vaccine IMA950 | NCIT_C94211 | [A cancer vaccine comprised of 11 peptides associated with glioblastoma multiforme (GBM), with potential immunomodulating and antineoplastic activities. Vaccination with glioblastoma multiforme multi-antigen vaccine IMA950 stimulates the host immune system to mount a cytotoxic T-lymphocyte (CTL) response as well as a T-helper (Th) immune response against tumor cells expressing these peptides, potentially resulting in decreased tumor growth of GBM. Peptides in IMA950 consist of the following: brevican (BCAN); chondroitin sulfate proteoglycan 4 (CSPG4); fatty acid binding protein 7, brain (FABP7); insulin-like growth factor 2 mRNA binding protein 3 (IGF2BP3); neuroligin 4, X-linked (NLGN4X); neuronal cell adhesion molecule (NRCAM); protein tyrosine phosphatase, receptor-type, Z polypeptide 1 (PTPRZ1); tenascin C (TNC); Met proto-oncogene (MET); baculoviral IAP repeat-containing 5 (BIRC5); and hepatitis B virus core antigen.] |
| Peptide Vaccine | NCIT_C1752 | [A vaccine based on a synthetic peptide or peptides produced from tissue samples or recombinant sources. Vaccination with peptide vaccines may elicit host immune responses against antigen-expressing cells.] |
| Number of Frames | NCIT_C69267 | [The total number of frames in a multiframe image.] |
| Number | NCIT_C25337 | [A numeral or string of numerals expressing value, quantity, or identification.] |
| Fluorodeoxygalactose F-18 | NCIT_C94212 | [A radioconjugate containing the D-galactose analogue 2-deoxy-2-fluoro-D-galactose (FDGal) labeled with the positron-emitting radioactive isotope fluorine F18, used for imaging upon positron emitting tomography (PET). With cell uptake much higher in tumor cells compared to normal cells, the F18 moiety of fluorodeoxygalactose F-18 can be visualized upon PET imaging and this agent can be used as a tracer for the evaluation of galactose tumor uptake and metabolism.] |
| Image Type | NCIT_C69268 | [A category of image.] |
| Type | NCIT_C25284 | [Something distinguishable as an identifiable class based on common qualities.] |
| Choline Kinase Alpha Inhibitor TCD-717 | NCIT_C94213 | [A small-molecule inhibitor of choline kinase alpha (CHKA), with potential antineoplastic activity. TCD-717 targets and binds to CHKA, an enzyme that plays a key role in the synthesis of phosphatidylcholine, the major phospholipid in eukaryotic cell membranes. Blockade of this enzyme induces cells to activate a different route for phospholipid production which causes a toxic effect and eventually leads to cell destruction. CHKA, overexpressed in human cancer cells while only minimally expressed in normal cells, appears to play a significant role in cellular proliferation, evasion of apoptosis, increased cell motility and metastasis.] |