All terms in NCIT
| Label | Id | Description |
|---|---|---|
| Sargramostim/Zidovudine | NCIT_C15480 | |
| Androgen Therapy | NCIT_C15482 | [Administration of male hormones to produce a clinical effect.] |
| Progestin Therapy | NCIT_C15484 | [The use of progestogens in the treatment of any disease or disorder.] |
| Childhood Lymphocyte-Rich Classic Hodgkin Lymphoma | NCIT_C7054 | [Lymphocyte-rich classic Hodgkin lymphoma occurring in childhood.] |
| Neurotoxicity Attenuation | NCIT_C15485 | |
| Precursor Lymphoid Neoplasm | NCIT_C7055 | [A neoplasm of immature malignant lymphocytes (lymphoblasts) committed to the B-cell or T-cell lineage. Neoplasms involving the bone marrow and the peripheral blood are called precursor lymphoblastic leukemias or acute lymphoblastic leukemias. Neoplasms involving primarily lymph nodes or extranodal sites are called lymphoblastic lymphomas. -- 2003] |
| Prostaglandin Inhibition | NCIT_C15486 | |
| Hypercalcemia Therapy | NCIT_C15487 | [Any care or treatment provided for the resolution of an abnormally high blood calcium concentration.] |
| Disease, Disorder or Finding | NCIT_C7057 | [A condition that is relevant to human neoplasms and non-neoplastic disorders. This includes observations, test results, history and other concepts relevant to the characterization of human pathologic conditions.] |
| Drug Modulation | NCIT_C15488 | |
| Meningioma by Morphology | NCIT_C7050 | |
| Releasing Hormone Agonist Therapy | NCIT_C15489 | [The use of releasing hormone agonists to bind to specific receptors and ultimately downregulate the activity of its specific hormone.] |
| Meningioma by Site | NCIT_C7051 | |
| Meningioma | NCIT_C3230 | [A generally slow growing tumor attached to the dura mater. It is composed of neoplastic meningothelial (arachnoidal) cells. It typically occurs in adults, often women and it has a wide range of histopathological appearances. Of the various subtypes, meningothelial, fibrous and transitional meningiomas are the most common. Most meningiomas are WHO grade I tumors, and some are WHO grade II or III tumors. Most subtypes share a common clinical behavior, although some subtypes are more likely to recur and follow a more aggressive clinical course. (Adapted from WHO)] |
| NF2 Gene Inactivation | NCIT_C36686 | |
| Loss of Chromosome 1p | NCIT_C36501 | [A cytogenetic abnormality that refers to the loss of all or part of the short arm of chromosome 1 (1p). It has been described in gliomas, meningiomas, neuroblastomas, hepatocellular carcinomas, breast carcinomas, acinar prostate adenocarcinomas, pancreatic carcinomas, and adrenal gland adenomas.] |
| Gain of Chromosome 17q | NCIT_C36484 | [A cytogenetic abnormality that refers to the duplication of all or part of the long arm of chromosome 17.] |
| Gain of Chromosome 9q | NCIT_C36483 | |
| Gain of Chromosome 12q | NCIT_C36441 | [A chromosomal aberration where there are 3 copies of the long arm of chromosome 12. This aberration is associated with 12q duplication syndrome.] |
| Gain of Chromosome 20q | NCIT_C36480 |