All terms in MESH
| Label | Id | Description |
|---|---|---|
| Non-Medical Public and Private Facilities | D000072182 | [Spaces and needed equipment provided for a specific, non-medical function or service used by the public or specific group of persons.] |
| C3H-1 protein, Xenopus | C118206 | |
| allergen Ara h3 | C118207 | |
| ZFR protein, mouse | C118208 | |
| Deafness, Autosomal Dominant 49 | C564250 | |
| N-(3-carboxyphenyl)anthranilic acid | C118209 | |
| Orofacial Cleft 4 | C564251 | |
| Spumavirus | D016092 | [Genus of non-oncogenic retroviruses which establish persistent infections in many animal species but are considered non-pathogenic. Its species have been isolated from primates (including humans), cattle, cats, hamsters, horses, and sea lions. Spumaviruses have a foamy or lace-like appearance and are often accompanied by syncytium formation. SIMIAN FOAMY VIRUS is the type species.] |
| Mason-Pfizer monkey virus | D016093 | [A species of BETARETROVIRUS isolated from mammary carcinoma in rhesus monkeys. It appears to have evolved from a recombination between a murine B oncovirus and a primate C oncovirus related to the baboon endogenous virus. Several serologically distinct strains exist. MPMV induces SIMIAN AIDS.] |
| Betaretrovirus | D030121 | [Previously a genus of the family RETROVIRIDAE consisting of oncogenic exogenous retroviruses that contain type D particles. It included the MASON-PFIZER MONKEY VIRUS. Now it represents one of the two kinds of viruses in the genus BETARETROVIRUS. The core of the type D viruses is the intracytoplasmic type A particle and its mechanism of budding from the cell membrane is similar to the type B oncoviruses., A genus of the family RETROVIRIDAE consisting of viruses with either type B or type D morphology. This includes a few exogenous, vertically transmitted and endogenous viruses of mice (type B) and some primate and sheep viruses (type D). MAMMARY TUMOR VIRUS, MOUSE is the type species., Previously a genus of the family RETROVIRIDAE consisting primarily of a few exogenous, vertically transmitted and endogenous viruses of mice. Now it represents one of the two kinds of viruses in the genus BETARETROVIRUS.] |
| Retroviruses, Simian | D015301 | [Classes of retroviruses for which monkeys or apes are hosts. Those isolated from the West African green monkey and the Asian rhesus macaque monkey are of particular interest because of their similarities to viruses causing cancer and acquired immunodeficiency syndrome (AIDS) in humans.] |
| Simian Acquired Immunodeficiency Syndrome | D016097 | [Acquired defect of cellular immunity that occurs naturally in macaques infected with SRV serotypes, experimentally in monkeys inoculated with SRV or MASON-PFIZER MONKEY VIRUS; (MPMV), or in monkeys infected with SIMIAN IMMUNODEFICIENCY VIRUS.] |
| Slow Virus Diseases | D012897 | [Diseases of viral origin, characterized by incubation periods of months to years, insidious onset of clinical manifestations, and protracted clinical course. Though the disease process is protracted, viral multiplication may not be unusually slow. Conventional viruses produce slow virus diseases such as SUBACUTE SCLEROSING PANENCEPHALITIS, progressive multifocal leukoencephalopathy (LEUKOENCEPHALOPATHY, PROGRESSIVE MULTIFOCAL), and AIDS. Diseases produced by unconventional agents were originally considered part of this group. They are now called PRION DISEASES.] |
| Lentivirus Infections | D016180 | [Virus diseases caused by the Lentivirus genus. They are multi-organ diseases characterized by long incubation periods and persistent infection.] |
| Monkey Diseases | D008992 | [Diseases of Old World and New World monkeys. This term includes diseases of baboons but not of chimpanzees or gorillas (= APE DISEASES).] |
| Gerstmann-Straussler-Scheinker Disease | D016098 | [An autosomal dominant familial prion disease with a wide spectrum of clinical presentations including ATAXIA, spastic paraparesis, extrapyramidal signs, and DEMENTIA. Clinical onset is in the third to sixth decade of life and the mean duration of illness prior to death is five years. Several kindreds with variable clinical and pathologic features have been described. Pathologic features include cerebral prion protein amyloidosis, and spongiform or neurofibrillary degeneration. (From Brain Pathol 1998 Jul;8(3):499-513; Brain Pathol 1995 Jan;5(1):61-75)] |
| Prion Diseases | D017096 | [A group of genetic, infectious, or sporadic degenerative human and animal nervous system disorders associated with abnormal PRIONS. These diseases are characterized by conversion of the normal prion protein to an abnormal configuration via a post-translational process. In humans, these conditions generally feature DEMENTIA; ATAXIA; and a fatal outcome. Pathologic features include a spongiform encephalopathy without evidence of inflammation. The older literature occasionally refers to these as unconventional SLOW VIRUS DISEASES. (From Proc Natl Acad Sci USA 1998 Nov 10;95(23):13363-83)] |
| Heredodegenerative Disorders, Nervous System | D020271 | [Inherited disorders characterized by progressive atrophy and dysfunction of anatomically or physiologically related neurologic systems.] |
| Electronic Prescribing | D055695 | [The use of COMPUTER COMMUNICATION NETWORKS to store and transmit medical PRESCRIPTIONS.] |
| Prescriptions | D055656 | [Directions written for the obtaining and use of PHARMACEUTICAL PREPARATIONS; MEDICAL DEVICES; corrective LENSES; and a variety of other medical remedies.] |